Omeprazole
Clinical Particulars
Omeprazole is a substituted benzimidazole
Pharmacokinetics
Mechanism of Action
Proton Pump Inhibitor: Omeprazole is a selective and irreversible proton pump inhibitor. In an acidic environment, activated omeprazole becomes a sulfenamide derivative that binds irreversibly at the secretory surface of parietal cells to the enzyme H+/K+ ATPase. This binding inhibits the transport of hydrogen ions into the stomach by 90% to 99%. Omeprazole blocks/prevents histamine, acetylcholine and gastrin-induced acid secretion. Omeprazole blocks gastric acid secretion irrespective of the stimulus. Omeprazole is inactive at physiological pH and has a limited effect on systemic ATP-ase.
Applications
Antacid & Gastroprotection
GERD Reflux Oesophagitis
Helicobacteriosis
Gastrinoma
CSF Reduction
Pharmacodynamics
Metabolism
Hepatic: Omeprazole is metabolised extensively in the liver to multiple metabolites (typically Hydroxyomeprazole and Omeprazole sulfone). Significant hepatic dysfunction can reduce the first-pass effect of Omeprazole, increasing the systemically available drug and prolonging its duration of action.
Elimination
Mixed Urine and Faeco-Biliary Excretion: Mainly as metabolites. Metabolites are mainly excreted in urine, but some are excreted via the biliary route in faeces.
Precautions
Availability
Identifiers
Systematic [IUPAC] Name: 6-methoxy-2-[ (4-methoxy-3,5-dimethylpyridin-2-yl) methylsulfinyl]-1H-benzimidazole]. Omeprazole is a substituted benzimidazole.
Formula: C17-H19-N3-O3-S
Pharmacotherapeutic group: Proton Pump Inhibitor and Cytochrome P450 2C19 Inhibitor.
ATC Vet Code: QA02BC01
ATC Code: A02BC01
Evidence Base
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